Studying at the University of Verona

Here you can find information on the organisational aspects of the Programme, lecture timetables, learning activities and useful contact details for your time at the University, from enrolment to graduation.

Academic calendar

The academic calendar shows the deadlines and scheduled events that are relevant to students, teaching and technical-administrative staff of the University. Public holidays and University closures are also indicated. The academic year normally begins on 1 October each year and ends on 30 September of the following year.

Academic calendar

Course calendar

The Academic Calendar sets out the degree programme lecture and exam timetables, as well as the relevant university closure dates..

Definition of lesson periods
Period From To
I sem. Oct 2, 2017 Jan 31, 2018
II sem. Mar 1, 2018 Jun 15, 2018
Exam sessions
Session From To
Sessione invernale d'esame Feb 1, 2018 Feb 28, 2018
Sessione estiva d'esame Jun 18, 2018 Jul 31, 2018
Sessione autunnale d'esame Sep 3, 2018 Sep 28, 2018
Degree sessions
Session From To
Sessione di laurea estiva Jul 13, 2018 Jul 13, 2018
Sessione di laurea autunnale Oct 22, 2018 Oct 22, 2018
Sessione di laurea invernale Mar 15, 2019 Mar 15, 2019
Period From To
Christmas break Dec 22, 2017 Jan 7, 2018
Easter break Mar 30, 2018 Apr 3, 2018
Patron Saint Day May 21, 2018 May 21, 2018
VACANZE ESTIVE Aug 6, 2018 Aug 19, 2018

Exam calendar

Exam dates and rounds are managed by the relevant Science and Engineering Teaching and Student Services Unit.
To view all the exam sessions available, please use the Exam dashboard on ESSE3.
If you forgot your login details or have problems logging in, please contact the relevant IT HelpDesk, or check the login details recovery web page.

Exam calendar

Should you have any doubts or questions, please check the Enrolment FAQs

Academic staff


Assfalg Michael +39 045 802 7949

Astegno Alessandra 045802 7955

Avesani Linda +39 045 802 7839

Benini Anna 045 8027603

Bentivoglio Marina 045 8027158

Boaretti Marzia 045 8027661

Bossi Alessandra Maria 045 802 7946 (Studio) - 045 802 7833 (Laboratorio)

Cecconi Daniela +39 045 802 7056; Lab: +39 045 802 7087

Constantin Gabriela 045-8027102

Corbo Vincenzo + 39 0458126029

Decimo Ilaria 045 8027509

Delledonne Massimo 045 802 7962; Lab: 045 802 7058

Dominici Paola 045 802 7966; Lab: 045 802 7956-7086

Giorgetti Alejandro 045 802 7982

Krampera Mauro 0458124034

Laudanna Carlo 045-8027689

Liptak Zsuzsanna +39 045 802 7032

Lleo'Fernandez Maria Del Mar 045 8027194

Malerba Giovanni 045/8027685

Mazzariol Annarita 045 8027690

Montagnana Martina +39 045 812 6698

Monti Francesca 045 802 7910

Signoretto Caterina 045 802 7195

Tagliaro Franco 045 8124618-045 8124246

Turco Alberto 0458027189

Vitulo Nicola 0458027982

Study Plan

The Study Plan includes all modules, teaching and learning activities that each student will need to undertake during their time at the University. Please select your Study Plan based on your enrolment year.

Final exam

1° Year

One course to be chosen among the following
One course to be chosen among the following

2° Year

Final exam

Legend | Type of training activity (TTA)

TAF (Type of Educational Activity) All courses and activities are classified into different types of educational activities, indicated by a letter.

S Placements in companies, public or private institutions and professional associations

Teaching code





English en

Scientific Disciplinary Sector (SSD)



II sem. dal Mar 1, 2018 al Jun 15, 2018.

Learning outcomes

The aim of this course is to cover a few general mechanisms of cell damage, giving information useful to understand the development of molecular pathology towards cell, tissue and organism pathology. The course covers the most important examples of damage to sub-cellular structures, particularly to the plasma membrane, describing in some details examples of alterations of transport, adhesion and signal transduction by membrane receptors. Different type of cell necrosis, factors determining regeneration or repair of injured tissues and molecular and cellular basis of reparative reactions and their pathologic consequences are also illustrated.


- Introduction to mechanisms of disease: from molecular pathology to alteration of tissue and organism homeostasis.
- Adaptation to cell injury: autophagy, unfolded protein response, alterations in cell volume and number.
- Forms of cell necrosis: necrosis, apoptosis, necroptosis and nettosis.
- General aspect of tissue regeneration and repair: from the concept of labile, stable and permanent cells to recent aspects of stem cell biology.
- Causes of cell pathology: major causes and highlighting of reactive oxygen specie generation as a key cause of cell pathology. Alterations of specific genes as a cause of tumor emergence and the principal hallmarks of cancer cells.
- Alterations of mitochondrial functions as a mechanism of cell pathology: mitochondrial diseases, role of mitochondria in cell aging, mitochondria and induction of apoptosis.
- Alterations of cell membrane structure induced by bacterial toxins and general mechanism of bacterial toxin action.
- Alterations of membrane transport: examples of alterations of anion and cation transport; cystic fibrosis as an example of a monogenic disease causing multiple organ pathology; alterations of lipid transport due to ABC protein mutation (Tangier disease, sitosterolemia) and geneal aspects of lipoprotein metabolism; alterations of iron transport in chronic inflammation and emocromatosis.
- Alterations of LDL-receptor expression and function as an example of molecular pathology leading to organisms pathology: classification of lipoprotein receptors; familiar hypercholesterolemia as a genetic form of deficient expression/function of the LDL-receptor; mechanisms of regulation of LDL-receptor expression by cell cholesterol; from reduced LDL-receptor expression to atherosclerosis (role of modified LDL as a cause of development of the atherosclerotic plaque and cellular basis of atherosclerosis).
- Alterations of expression/function of adhesive receptors: general aspects of cell adhesion; integrins as the most widely expressed adhesion molecules implicated in cell-extracellular matrix interaction (classification and ligands); mechanisms of regulation of integrin adhesion (inside-out signaling).
- Adhesive interactions implicated in leukocyte recruitment into inflammatory sites: physiology and pathology.
- Adhesive interactions implicated in the function of platelets in hemostatic reaction and mechanisms of activation of platelets.
- The coagulation system, the soluble component of hemostatic reactions: mechanisms of activation and negative regulation.
- Deficit of hemostatic functions and the etiopathogenesis of thrombosis.
- Physiopathology of signal transduction: general aspects and modality of signal transduction by membrane surface receptors.
- Signal transduction by receptors with an intrinsic tyrosine kinase activity: the Ras and the PI3K pathways; downstream targets of PIP3; AKT in regulation of cell growth, survival and metabolism; receptor tyrosine kinase mutations in cancer; development of tyrosine kinase inhibitors.
- The insulin receptor: mechanisms of signal transduction; the concept of insulin resistance and alterations of insulin signal transduction in type 2 diabetes; obesity, inflammation and type 2 diabetes.
- Mechanisms of signal transduction by trimeric G protein-coupled receptors: regulation of trimeric G proteins; down-stream effectors of Galpha-GTP and beta/gamma subunit of trimeric G proteins.
- Activation of phosphoinositide-specific Phospholipases C and regulation of cytoplasmic calcium transients.
- Trimeric G protein-coupled receptors, PI3Kgamma and regulation of cell migration.
- Cytoplasmic tyrosine kinases as signal transducers: major classes of cytoplasmic tyrosine kinases; mechanisms of regulation of Src and Abl kinases; cytoplasmic tyrosine kinases in integrin outside-in signaling and regulation of cytoskeleton rearrangements and cell survival; cytoplasmic tyrosine kinases and signal transduction by immune receptors.
- Other mechanisms of signal transduction by surface receptors: signal transduction by cadherins; signal transduction by Toll-like receptors and pathogenesis of the septic shock.
- Pathology of contractile cells: muscular dystrophies; mechanisms of cardiac hypertrophy.

Reference texts
Author Title Publishing house Year ISBN Notes
Kumar V, Abbas AK, Aster JC Robbins Basic Pathology (Edizione 10) Elsevier 2018

Examination Methods

oral exam

Type D and Type F activities

Modules not yet included

Career prospects

Module/Programme news

News for students

There you will find information, resources and services useful during your time at the University (Student’s exam record, your study plan on ESSE3, Distance Learning courses, university email account, office forms, administrative procedures, etc.). You can log into MyUnivr with your GIA login details.


For schedules, administrative requirements and notices on graduation sessions, please refer to the Graduation Sessions - Science and Engineering service.

List of theses and work experience proposals

theses proposals Research area
Studio delle proprietà di luminescenza di lantanidi in matrici proteiche Synthetic Chemistry and Materials: Materials synthesis, structure-properties relations, functional and advanced materials, molecular architecture, organic chemistry - Colloid chemistry
Stampa 3D di nanocompositi polimerici luminescenti per applicazioni in Nanomedicina Synthetic Chemistry and Materials: Materials synthesis, structure-properties relations, functional and advanced materials, molecular architecture, organic chemistry - New materials: oxides, alloys, composite, organic-inorganic hybrid, nanoparticles
3D-bioprinting biofabrication laboratory Various topics
Organ on-a-chip Various topics


As stated in point 25 of the Teaching Regulations for the A.Y. 2021/2022, attendance is not mandatory. However, professors may require students to attend lectures for a minimum of hours in order to be able to take the module exam, in which case the methods that will be used to check attendance will be explained at the beginning of the module. 
Please refer to the Crisis Unit's latest updates for the mode of teaching.

Career management

Area riservata studenti