Studying at the University of Verona

Here you can find information on the organisational aspects of the Programme, lecture timetables, learning activities and useful contact details for your time at the University, from enrolment to graduation.

The Study Plan includes all modules, teaching and learning activities that each student will need to undertake during their time at the University.
Please select your Study Plan based on your enrollment year.

1° Year

ModulesCreditsTAFSSD
7
A/B
BIO/16 ,BIO/17 ,MED/48
6
A/B
BIO/09 ,MED/48
6
A/B
BIO/09 ,M-PSI/01
5
A/B
M-DEA/01 ,MED/36 ,MED/42 ,MED/48
5
A/B
MED/34 ,MED/48 ,M-PED/01
8
A/B
BIO/10 ,BIO/13 ,FIS/07 ,MED/01

3° Year  activated in the A.Y. 2019/2020

ModulesCreditsTAFSSD
7
A/B/C
MED/43 ,MED/44 ,M-PSI/06 ,SECS-P/10 ,SPS/07
6
B
MED/06 ,MED/09 ,MED/24 ,MED/48
Prova finale
7
E
-
ModulesCreditsTAFSSD
7
A/B
BIO/16 ,BIO/17 ,MED/48
6
A/B
BIO/09 ,MED/48
6
A/B
BIO/09 ,M-PSI/01
5
A/B
M-DEA/01 ,MED/36 ,MED/42 ,MED/48
5
A/B
MED/34 ,MED/48 ,M-PED/01
8
A/B
BIO/10 ,BIO/13 ,FIS/07 ,MED/01
activated in the A.Y. 2019/2020
ModulesCreditsTAFSSD
7
A/B/C
MED/43 ,MED/44 ,M-PSI/06 ,SECS-P/10 ,SPS/07
6
B
MED/06 ,MED/09 ,MED/24 ,MED/48
Prova finale
7
E
-

Legend | Type of training activity (TTA)

TAF (Type of Educational Activity) All courses and activities are classified into different types of educational activities, indicated by a letter.




S Placements in companies, public or private institutions and professional associations

Teaching code

4S02742

Credits

2

Language

Italian

Scientific Disciplinary Sector (SSD)

MED/05 - CLINICAL PATHOLOGY

Period

FISIO VI 2^ ANNO - 1^ SEMESTRE dal Oct 1, 2018 al Nov 23, 2018.

Location

VICENZA

To show the organization of the course that includes this module, follow this link:  Course organization

Learning outcomes

At the end of the course the student is expected to address the following subjects:

• Etiopathogenesis of cell damage
• Causes of alteration of cell function (chemical, physical and biological)
• Etiopathogenesis of disease. Concept of homeostasis and its regulation
• Acute and chronic inflammation
• Immune system: cell components, their function and interaction
• Homeostasis of the immune response, transfusion and transplants
• Molecular and cellular basis of neoplastic transformation

Program


Introduction to pathology
• Cell homeostasis and alterations at the cell and organism levels
• The concept of biochemical lesion and subcellular localization
• Alteration of cell homeostasis: adaptation, necrosis, apoptosis, restoration of cell damage
• Examples of cell adaptation: alterations of cells status and differentiation

Causes of disease and mechanisms of cell damage
• General classification of the causes of disease: genetic (example: cystic fibrosis), environmental, multifactorial (example: atherosclerosis), chemical (example: alcohol), radiation, free radicals
• Mechanisms of healing

Oedema: pathogenesis and classification



Introduction to immunology and immunological damage

• Innate and acquired immunity: description of leukocytes involved and general mechanisms
• General features: antigen presentation, lymphocyte subclasses, complement
• MHC ad transplants
• Blood groups
• Mechanisms of immuological damage: anafilaxis, complement activation, role of antibodies and Fc receptor, cell-dependent cytotoxicity

Inflammation
• Role of blood vessels in inflammation
• Cells involved in inflammation
• Classification of inflammatory response based on prevalent features: erithematous, serous, fibrinous, purulent, necrotic-haemorragic
• Differences among acute and chronic inflammation
• Cytokines and their role in the regulation of the inflammatory response



Neoplasia

• Differences among benign and malign neoplasms
• Properties of neoplastic cells: growth, invasion and differentiation
• General classification: epithelial, mesenchimal, neuroectodermal and germinal, hemopoietc cells neoplasms
• Natural history of neoplasia: precancerous lesions, carcinoma in situ, metastasis. Preferential localization. TNM score.
• Molecular basis of neoplastic transformation: oncogenes and tumor suppressor genes. Mechanisms of activation/inhibition.
• Cancer etiology: physical, chemical and biological
• Mechanism of damage: compression, paraneoplastic syndromes, cachexia
• Cancer biomarkers and their clinical relevance

Reference texts
Author Title Publishing house Year ISBN Notes
Albi, E., Amberi-Impiombato, F.S., Curcio F., Moncharmont, B., Palese, A. Le basi cellulari e molecolari delle malattie (Edizione 2019) Sorbona Idelson Gnocchi 2018 ISBN 978-88-7947-671-3
Giovanni M. Pontieri Patologia generale & fisiopatologia generale. Per i corsi di laurea in professioni sanitarie (Edizione 3) Piccin 2018 8829921424

Examination Methods

Quiz: multiple choices. Oral test at request or if deemed appropriate by the commission.

Students with disabilities or specific learning disorders (SLD), who intend to request the adaptation of the exam, must follow the instructions given HERE