Studying at the University of Verona

Here you can find information on the organisational aspects of the Programme, lecture timetables, learning activities and useful contact details for your time at the University, from enrolment to graduation.

The Study Plan includes all modules, teaching and learning activities that each student will need to undertake during their time at the University.
Please select your Study Plan based on your enrollment year.

1° Year

ModulesCreditsTAFSSD
8
A/B
BIO/09 ,BIO/16 ,BIO/17 ,MED/46
7
A/B
BIO/13 ,MED/03 ,MED/07 ,MED/46
7
A/B
FIS/07 ,ING-INF/06 ,ING-INF/07 ,MED/01

2° Year  activated in the A.Y. 2016/2017

ModulesCreditsTAFSSD
4
B
MED/06 ,MED/11 ,MED/13 ,MED/15
4
A/B
BIO/14 ,MED/46

3° Year  activated in the A.Y. 2017/2018

ModulesCreditsTAFSSD
6
A/B
IUS/07 ,MED/42 ,M-PSI/01 ,SECS-P/07 ,SPS/07
Prova finale
7
E
-
activated in the A.Y. 2016/2017
ModulesCreditsTAFSSD
4
B
MED/06 ,MED/11 ,MED/13 ,MED/15
4
A/B
BIO/14 ,MED/46

Legend | Type of training activity (TTA)

TAF (Type of Educational Activity) All courses and activities are classified into different types of educational activities, indicated by a letter.




S Placements in companies, public or private institutions and professional associations

Teaching code

4S000336

Credits

2

Language

Italian

Scientific Disciplinary Sector (SSD)

MED/04 - EXPERIMENTAL MEDICINE AND PATHOPHYSIOLOGY

Period

TEC LAB BIOM LEZ 2A 1S dal Oct 3, 2016 al Nov 30, 2016.

To show the organization of the course that includes this module, follow this link:  Course organization

Learning outcomes

Knowledge of the main mechanisms of molecular and cellular injury and the basic reactions of cecells and tissues to the abnormal stimuli that underlie diseases.

Program

Molecular pathology
Chromosomal disorders
Mendelian single gene disorders
Complex multigenic disorders

Cellular responses to stress and toxic insults
Adaptation of cellular growth and differentiation (hyperplasia, hypertrophy, hypotrophy, metaplasia, dysplasia)
Causes of cell injury (hypoxia, free radicals, bacterial- and viral-induced cell injury)
Cell death: necrosis and apoptosis

Inflammation
Acute inflammation:
Reaction of blood vessels in acute inflammation
Reaction of leukocytes in inflammation (chemotaxis, phagocytosis and killing)
Cells involved in acute inflammation
Mediators of inflammation from cells and plasma
Systemic effects of acute inflammation
Outcomes of acute inflammation
Chronic inflammation
Causes
Morphological features
Role of macrophages
Chronic hepatitis model
Granulomatous inflammation model

Blood vessel injury
Atherosclerosis

Examination Methods

Oral test

Students with disabilities or specific learning disorders (SLD), who intend to request the adaptation of the exam, must follow the instructions given HERE